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Gene | CHEK2 |
Variant | T367Mfs*15 |
Impact List | frameshift |
Protein Effect | loss of function |
Gene Variant Descriptions | CHEK2 T367Mfs*15 indicates a shift in the reading frame starting at amino acid 367 and terminating 15 residues downstream causing a premature truncation of the 543 amino acid Chek2 protein (UniProt.org). T367Mfs*15 results in reduced tyrosine phosphorylation in response to DNA damage (PMID: 16982735), loss of Kap1 phosphorylation at serine (S)-473 compared to wild-type Chek2 in in vitro assays and in cell culture (PMID: 31050813, PMID: 34903604). |
Associated Drug Resistance | |
Category Variants Paths |
CHEK2 mutant CHEK2 inact mut CHEK2 T367fs CHEK2 T367Mfs*15 |
Transcript | NM_007194.4 |
gDNA | chr22:g.28695869delG |
cDNA | c.1100delC |
Protein | p.T367Mfs*15 |
Source Database | RefSeq |
Genome Build | GRCh38/hg38 |
Transcript | gDNA | cDNA | Protein | Source Database | Genome Build |
---|---|---|---|---|---|
NM_007194.3 | chr22:g.28695869delG | c.1100delC | p.T367Mfs*15 | RefSeq | GRCh38/hg38 |
NM_007194.4 | chr22:g.28695869delG | c.1100delC | p.T367Mfs*15 | RefSeq | GRCh38/hg38 |
NM_007194 | chr22:g.28695869delG | c.1100delC | p.T367Mfs*15 | RefSeq | GRCh38/hg38 |
Clinical Trial | Phase | Therapies | Title | Recruitment Status | Covered Countries | Other Countries |
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